A new study demonstrates that long-term oral nicotine administration reprograms aging-related metabolism and slows motor decline in mice. By targeting the sphingolipid-energy axis, nicotine improved energy homeostasis, preserved gut microbial diversity, and supported neuromuscular resilience.
A new study demonstrates that long-term oral nicotine administration reprograms aging-related metabolism and slows motor decline in mice. By targeting the sphingolipid-energy axis, nicotine improved energy homeostasis, preserved gut microbial diversity, and supported neuromuscular resilience.
Share this Article
Recommended